Cordelia Schiene‐Fischer

Max Planck Research Unit for Enzymology of Protein Folding

Papers

1

Total Citations

39

H-Index

1

About

Cordelia Schiene‐Fischer is a leading biochemist whose research centers on peptidylprolyl cis-trans isomerases (PPIases), particularly cyclophilins, and their roles in protein folding, cellular signaling, and disease. Her most cited work, "Secreted Cyclophilin A Mediates Matrix Assembly of Hensin" (2008, 39 citations), reveals a groundbreaking mechanism: extracellular cyclophilin A acts as a chaperone to assemble the multidomain protein hensin (DMBT1 ortholog) into the extracellular matrix, driving terminal epithelial differentiation. This discovery bridges protein isomerase activity with tissue morphogenesis and innate immunity, highlighting cyclophilin A’s dual intracellular and extracellular functions. Schiene‐Fischer’s contributions have illuminated how PPIases regulate protein conformation beyond the ribosome, impacting fields from cancer biology to fibrosis. Her work is distinguished by its focus on secreted cyclophilins as mediators of matrix assembly and differentiation, earning recognition for its translational potential. With a citation record that underscores her influence, she continues to unravel the molecular logic of isomerases in health and disease, offering students a compelling model of how fundamental protein chemistry drives complex physiological outcomes.

Research Focus

Key Achievements

1
H-Index
1
Papers
39
Total Citations
39
Avg Citations/Paper
🏆 Most Cited Paper
Secreted Cyclophilin A, a Peptidylprolyl cis-trans Isomerase, Mediates Matrix Assembly of Hensin, a Protein Implicated in Epithelial Differentiation
39 citations · 2008
📈 Most Prolific Year: 2008 (1 Papers)
🤝 Key Collaborators: 8
🏛 Institutions: Max Planck Research Unit for Enzymology of Protein Folding

Top Papers

  1. 1

Key Collaborators

Contact & Links

Available for collaboration
Content generated · 15 days ago